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5 Reasons Not To Take Anti-Depressant Drugs By Lennard Davis Reason One:
A study in the Journal of the
American Medical Association says that SSRI's like Paxil and Prozac are no
more effective in treating depression than a placebo pill. That means they
are 33 per cent effective, which is the percent of patients who will respond
well to a sugar pill. The article goes on to say that although SSRI's are
effective to some degree in treating severe depression they don't have any
effect on the routine type of depressions they are most often used to treat.
The take-home message is--don't take SSRI's if you have normal, mild, or
routine depression. It's a waste of money, and the drugs have serious
side-effects including loss of sexual drive. Reason Two:
A January 4 article in MedPage
Today cites a study done at Columbia University and Johns Hopkins. The study
says that doctors routinely prescribe not one but two or three SSRI's and
other psychopharmological drugs in combination with few if any serious
studies to back up the multiple usage. It's pretty obvious that the reason
for these multiple prescriptions is that if one drug doesn't work, then
perhaps two or three will. Doctors are in essence performing uncontrolled
experiments on their patients, hoping that in some scattershot way they might
hit on a solution. But of course drugs have dangerous interactions and most
physicians are shooting in the dark with all the dangers that attend such bad
marksmanship. Reason Three:
More and more psychiatric
disorders are appearing that might be called "lifestyle" diseases.
What was called shyness, sadness, restlessness, shopping too much, high sex
drive, low sex drive, and so on have increasingly been seen as diseases and
many more will appear in the new DSM, the diagnotstic manual of psychological
and psychiatric disorders. Increasingly the criteria for inclusion in the DSM
involves whether the disorder responds to a category of drugs. If, as we've
just seen, one of the key class of drugs that for 20 years has been
considered effective now fails, what does that say for this idea that if a
disease responds to a particular drug, then it is a particular disease? We
have to rethink the whole biological basis for lifestyle disorders. Reason Four:
We're an over-medicated
society, and the goal of drug companies and a compliant and harried medical
establishment is ultimately to have some drug coursing through every
individuals bloodstream. It's a lot easier to quickly pop a pill or prescribe
than it is to explore the reasons for a person's distress. Many of us
remember the scenario in 1960's science fiction movies of a dystopic future
or Soviet-style world with drugs used to control minds. Well, that future is
here and the social control we dreaded is now accepted in the form of a pill. Reason Five:
The whole serotonin hypothesis
is challenged by these findings. What this new information shows is that
there may be some help using SSRIs if there is a severe shortage of
serotonin, but the average person's depression cannot simply be related to a
"chemical imbalance." The human brain is too complicated and so are
we to have a simple, quick explanation related to seratonin alone. We have no
way of measuring serotonin the brain of a living person, short of cutting
open the skull. We have not come up with what a normal level of serotonin
should be and below which we can say that you would be depressed and above
which we can say you will be happy. People with high serotonin levels can be
depressed and those with low levels can be happy. Serotonin inducing drugs
like ecstasy can make you feel very happy, but so can alcohol and heroin. We
have to go back to the drawing boards on this one, so don't ever let anyone
say "I've got a chemical imbalance" without asking them what they
actually mean and where is the science to prove that statement. _________________________________________________________________________________________________ Copyright
by Lennard Davis Source: www.psychologytoday.com |
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